Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

A theoretical model based upon a set of chemical reaction equations was established. It was assumed that activated LFA-1 cannot resume in passive or low affinity state as long as it is bound to ICAM-1. The initial results of the model suggest a role of active cellular processes (inside-out signaling) in LFA-1 activation.

Type

Journal article

Journal

Annals of Biomedical Engineering

Publication Date

01/12/2000

Volume

28